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Sel- sel darah tak dapat melewati dinding kapiler. Filtrate (hasil filtrasi) dikumpulkan glomerular capsule, kemudian ke renal tubule(Reabsorpsi) Reabsorption. Peritubular capillaries menyerap : Air. Glucosa. Asam amino. Ion- ion. Penyerapan sebagian besar terjadi pada proximal convoluted tubule. Zat yang tak direabsorbsi. Sisa metabolisme Nitrogenous. Air yang berlebihan. Sekresi (Secretion)(Kebalikan Reabsorpsi)Beberapa zat berpindah dari peritubular capillaries ke renal tubules. Askep Karsinoma Nasofaring downloads at Ebook-kings.com - Download free pdf files,ebooks and documents - Vascular Endothelial Growth Factor pada Karsinoma.Ii LEMBAR PENGESAHAN JURNAL MEDIA MEDIKA MUDA KTI FAKTOR RISIKO KOLONISASI Klebsiella sp. PADA NASOFARING BALITA (Penelitian belah lintang pada balita yang tinggal di. Hydrogen and potassium ions. Creatinine. Zat- zat meninggalkan renal tubule berpindah ke ureter. Karakteristik Urine digunakan untuk Diagnosa. Warna kuning disebabkan pigment urochrome (dari penghancuran hemoglobin) and zat- zat terlarut lain. Steril. Sedikit beraromap. H Normal sekitar 6 (4. Specific gravity : 1. WHO 3 menujukkan respon. Penelitian ini bertujuan untuk. CT- scan pasien karsinoma nasofaring, pada. Metode: Penelitian ini. Kariadi,Semarang pada 1 Januari 2. Desember. 2. 00. 9. Uji hipotesis yang digunakan ialah uji komparatif Chi- square (X2) dan Fisher’s. Exact. Hasil: Terdapat 5. Pada stadium II dan III. WHO 3 dan 5. 0% pasien WHO 2 menunjukan respon positif (respon. Hasil dari uji dengan Fisher’s Exact pada stadium II dan III, p=0. Pada stadium IV, 6. WHO 2 dan 6. 1. 1% WHO 3. Hasil tes Chi. square pada stadium IV, p=0. Kesimpulan: Terdapat hubungan. CT scan pada pasien dengan karsinoma nasofaring pada. II dan III. Tidak terdapat hubungan bermakna antara klasifikasi. CT scan. pada pasien dengan karsinoma nasofaring pada stadium IV. BAB IPENDAHULUAN1. LATAR BELAKANGKarsinoma nasofaring (KNF) merupakan. Faktor etnik dan. Insiden KNF yang berbeda secara. Epstein- Barr. Secara global, diperkirakan. Insiden kanker ini cukup jarang di beberapa negara, yakni. Insiden KNF di Amerika 1- 2 kasus per 1. Namun tumor ini sangat banyak. Cina. Asia Tenggara, Afrika Utara dan daerah Arctic. Insiden KNF tertinggi di dunia. Cina bagian Selatan khususnya suku Kanton di. Guang Dong dan daerah Guangxi dengan angka mencapai lebih dari 5. Indonesia termasuk salah satu Negara dengan prevalensi. KNF yang termasuk tinggi di luar Cina. Data registrasi kanker di. Indonesia berdasarkan histopatologi tahun 2. KNF menempati. urutan pertama dari semua tumor ganas primer pada laki – laki dan urutan ke 8. Dari keseluruhan KNF tersebut, proporsi KNF subtype nonkeratinizing. WHO- 2) dan undifferentiated carcinoma (WHO- 3) adalah sama. Karsinoma nasofaring lebih sering pada. Kanker ini dapat mengenai semua umur dengan. RUMUSAN MASALAHBerdasarkan. TUJUAN1. 3. 1. Tujuan. Tujuan. khusus. 1. Untuk mengetahui distribusi umur pada penderita karsinoma nasofaring . Untuk mengetahui distribusi jenis kelamin pada penderita karsinoma nasofaring . Untuk mengetahui distribusi. Untuk mengetahui distribusi stadium pada penderita karsinoma nasofaring . Untuk mengetahui distribusi. Untuk mengetahui distribusi tipe histopatalogis pada penderita karsinoma nasofaring 1. METODEDikumpulkan data sekunder hasil. Hubungan Ekspresi Latent Membrane Protein- 1 Virus. Epstein- Barr dengan Subtipe Karsinoma Nasofaring), yaitu berupa identitas. LMP- 1. Data untuk penelitian tersebut merupakan data sekunder. KNF Januari 2. 00. Juni 2. 01. 0 dari laboratorium PA FK. Dr. M. Djamil Padang dan RSUD. Achmad Muchtar Bukittinggi. Selanjutnya dari blok. Deparafinisasi. dilakukan dengan xylol dan rehidrasi dengan alkohol. Pulasan imunohistokimia. Dako). Ekspresi p. Nilai p < 0,0. HASILSampel terdiri dari 5. Berdasarkan jenis kelamin, jumlah pasien. Berdasarkan umur, jumlah. Jumlah. pasien yang terbanyak datang pada stadium IV, yaitu 3. Menurut. klasifikasi histopatologis, pasien terbanyak didiagnosisdengan WHO tipe 3 (undifferenciated. WHO tipe 1 (keratinizing squamous cell carcinoma). Sampel dalam. penelitian ini terbagi menjadi dua berdasarkan stadium kliniknya. Berdasar. stadium klinik, terdapat 2. II dan III. serta 3. IV. Respon. kemoradiasi berdasarkan gambaran CT- Scan pasien karsinoma nasofaring, disajikan. Karsinoma. nasofaring merupakan tumor ganas daerah kepala dan leher yang terbanyak. Indonesia. Insiden KNF yang berbeda secara. Epstein- Barr. 3 Secara. Insiden kanker ini cukup jarang. Insiden KNF di. Amerika 1- 2 kasus per 1. Dari keseluruhan KNF tersebut, proporsi KNF. WHO- 2) dan undifferentiated. WHO- 3) adalah sama banyak, yaitu masing- masing 3. Karsinoma. nasofaring lebih sering pada laki- laki dibanding perempuan. Kanker ini dapat. Kasus KNF juga pernah dilaporkan terjadi pada. Sayang sekali tumor ganas ini tidak mempunyai. Bahkan pada lebih dari. KNF. Berdasarkan klasifikasi histologi WHO tahun 1. KNF dibagi. menjadi tiga subtipe yaitu; squamous cell carcinoma (WHO- 1), nonkeratinizing. WHO- 2) dan undifferentiated carcinoma (WHO- 3). Undifferentiated. WHO- 3) merupakan subtipe histologi yang utama di daerah endemik. WHO- 1 jarang (< 5%). Terdapat tiga faktor etiologi utama yang. KNF yaitu infeksi EBV, kerentanan genetic dan faktor lingkungan. Di daerah endemik, infeksi EBV terutama berkaitan dengan KNF subtipe WHO- 2 dan. WHO- 3, sedangkan untuk subtipe WHO- 1 masih menjadi perdebatan. Virus. Epstein- Barr yang ditransmisikan melalui saliva yang terinfeksi ke tempat. Hal. ini membuat sel yang terinfeksi menjadi immortal melalui induksi transformasi. Infeksi EBV yang laten dan persisten tersebut pada. KNF menunjukkan pola laten tipe II yang ditandai dengan ekspresi EBV nuclear. EBNA) - 1,latent membrane protein (LMP) - 1, 2 dan EBVencoded. RNA (EBER). 3,1. 5 LMP- 1 merupakan gen laten EBV yang pertama ditemukan yang. LMP- 1 merupakan onkogen virus yang mirip reseptor permukaan sel. EBV dari apoptosis dengan menginduksi. BCL- 2, A2. 0 dan MCL- 1. LMP- 1 juga terlibat dalam. G1/S) dan inhibisi. Gen p. 53 merupakan salah satu dari gen supresor tumor. Gen ini. mendeteksi kerusakan DNA,membantu perbaikan DNA melalui penghentian fase G1. DNA. Sel yang mengalami. DNA dan tidak dapat diperbaiki diarahkan oleh p. Hampir lebih dari 5. Fungsi gen p. 53 dapat diinaktivasi oleh berbagai mekanisme. DNA tertentu. Salah satu virus DNA tersebut. EBV. Virus Epstein- Barr dapat mengikat protein p. Penelitian imunohistokimia memperlihatkan bahwa infeksi EBV. KNF berhubungan dengan akumulasi protein p. BCL- 2. Namun. penelitian lain menyimpulkan bahwa EBV merupakan faktor etiologi yang penting. BCL- 2. Perkembangan KNF melibatkan. Namun mekanisme inhibisi supresor tumor ini, unik. KNF. Pada kebanyakan kanker kepala dan leher, kadar p. Namun, p. 53 pada KNF tidak mengikuti pola klasik ini. Tidak. adanya mutasi p. KNF memberi kesan bahwa suatu protein virus dapat. Dari suatu penelitian didapatkan bahwa sel- sel KNF. LMP- 1 yang tinggi berkorelasi. Mutasi p. 53 relatif jarang pada KNF. Wild type p. 53. ini gagal menginduksi apoptosis pada KNF. Pemeriksaan ekspresi LMP- 1 dan p. IHK) dari jaringan tumor KNF. Pada. penelitian ini ingin dibuktikan korelasi antara ekspresi Latent Membrane. Protein - 1 virus Epstein- Barr dengan ekspresi p. Diduga. disebabkan karena mereka memakan makanan yang diawetkan dalam musim dingin. Selain itu faktor. Tetapi sudah hampir dapat dipastikan bahwa. Epstein- barr, karena pada semua. EEB yang cukup tinggi (Efiaty& Nurbaiti,2. TANDA DAN GEJALA Berkait dengan hal tersebut, maka gejala yang timbul pada. Gejala nasofaring. Gejala. nasofaring dapat berupa epistaksis ringan atau sumbatan hidung. Hal ini perlu pemeriksaan. Gejala yang nampak dari gangguan tersebut adalah diplopia dan. Penderita. akan mengalami kesulitan dalam mrngunyah. PEMERIKSAAN PENUNJANGa. Nasofaringoskopib. Untuk diagnosis pasti ditegakkan dengan Biopsi. Pemeriksaan CT- Scan daerah kepala dan leher untuk mengetahui keberadaan tumor sehingga tumor. Pemeriksaan Serologi Ig. A anti EA dan Ig. A anti. VCA untuk mengetahui infeksi virus. E- B. e. Pengerokan dengan kuret daerah lateral. Efiaty& Nurbaiti,2. PENATALAKSANAAN MEDISa. Radioterapi. merupakan’pengobatan,utama. Pengobatan tambahan yang diberikan dapat berupa. Pemberian ajuvan. Cis- platinum, bleomycin dan 5- fluorouracil. Sedangkan. kemoterapi praradiasi dengan epirubicin dan cis- platinum. Kombinasi. kemo- radioterapi dengan mitomycin C dan 5- fluorouracil oral sebelum diberikan. KARAKTERISTIK SUBJEK PENELITIANPada penelitian ini didapatkan kelompok. KNF adalah pada usia 5. Usia insiden KNF. KNF mencapai puncaknya pada umur 4. Pola distribusi. usia KNF bervariasi pada berbagai daerah yang berbeda di dunia. Hirayama. melaporkan bahwa insiden pada kedua jenis kelamin dimulai setelah usia 2. Frekuensi KNF. yang relative tinggi ini diantara populasi muda merupakan gambaran khas di area. Uganda, Kenya, Sudan dan Tunisia. Hirayama juga. melaporkan bahwa bahwa usia puncak pada remaja jarang terjadi pada suku Cina di. Amerika. Balakrishnan et al. Penelitian KNF pada anak. Sahraoui et al., 1. Casablanca, didapatkan. WHO- 3 sebanyak 8. Peneliti lain juga pernah menemukan. KNF termuda yang berumur 4 tahun. Insiden KNF pada anak- anak mempunyai. Laki- laki merupakan. KNF terbanyak pada penelitian ini, dengan ratio laki- laki dan. Hasil yang didapatkan ini tidak jauh berbeda dengan. KNF lebih sering pada laki- laki daripada. Lebih banyaknya penderita. KNF pada laki- laki dibanding perempuan. Peneliti. terdahulu mendapatkan bahwa orang yang merokok selama sepuluh tahun atau lebih. KNF. 1. 9 Lin et al. Pada penelitian ini didapatkan KNF subtype WHO- 2. Di daerah endemik Cina, mayoritas KNF adalah. WHO- 2 dan WHO- 3. Sebaliknya, di daerah non endemic seperti Amerika. WHO- 1 merupakan subtipe yang terbanyak. Infeksi Epstein- Barr Virus berkaitan. WHO- 3 dan sebagian dengan subtype WHO- 2, namun tidak dengan. WHO- 1. 1. 3 Jadi kemungkinan besar infeksi EBV juga merupakan faktor. KNF pada penelitian ini. EKSPRESI LMP- 1 PADA KARSINOMA NASOFARINGPada penelitian ini, secara. LMP- 1 positif, yaitu pada 9. Hasil ini dapat menggambarkan bahwa infeksi EBV memang merupakan faktor. KNF ini, sama seperti daerah endemik lainnya. Dari. kepustakaan didapatkan bahwa LMP- 1 sering terdeteksi pada biopsi KNF tetapi. Berdasarkan laporan yang. KNF, LMP- 1. dapat divisualisasikan pada mayoritas sel- sel tumor dengan menggunakan teknik imunohistokimia. Demikian juga dengan Sheu et al.,2. Hasil yang hampir sama juga didapatkan oleh Suharto dkk,2. KNF. Penelitian yang dilakukan oleh. Taweevisit,2. 3 di Bangkok pada 6. KNF subtipe WHO- 3, mendapatkan hasil. Hal ini menunjukkan bahwa. KNF. Sebagian besar KNF. EBV. Hasil penelitian yang didapatkan oleh Shaoi et al.,2. KNF secara signifikan berkorelasi. LMP- 1. Penelitian ini mendapatkan hasil yang sama dengan. KNF mempunyai kadar p. LMP- 1 berkorelasi dengan semakin tingginya ekspresi. LMP- 1 menghambat pengaruh penekanan wild type p. LMP- 1 juga mampu mengalahkan hambatan. Disamping itu, LMP- 1 dapat. KNF. BAB IIIPENUTUP3. KESIMPULANKarsinoma Nasofaring merupakan. Cina bagian. selatan. Terdapat tiga faktor etiologi utama yang berhubungan dengan KNF yaitu infeksi. EBV, kerentanan genetik dan faktor lingkungan. 3 respuesta ante las intoxicaciones agudas por plaguicidas manual para el sanitario vigilancia epidemiol Reglamento sobre Condiciones Sanitarias y Ambientales b. SISTEMA DE VIGILANCIA EPIDEMIOLOGICA DE ERGONOMIA INFORME TECNICO DE LA EMPRESA . You can download the paper by clicking the button above. Departamento Salud Ocupacional de ISP, difundi Postgrado en Salud Ocupacional e Higiene del Ambiente Laboral. 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